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Open Journal Of Apoptosis

2017-06-29T13:42:01Z

Person8virgo: Створена сторінка: agents/materials/analysis tools: NCM CRC [http://www.ncbi.nlm.nih.gov/pubmed/ 23388095 23388095] KGS LF SS MicroRNAs in LVH DS MAC MEA AB NC UM LER. Wrote the...

agents/materials/analysis tools: NCM CRC [http://www.ncbi.nlm.nih.gov/pubmed/ 23388095 23388095] KGS LF SS MicroRNAs in LVH DS MAC MEA AB NC UM LER. Wrote the paper: NCM AB NC UM LER. References 1. Dorn GW 2nd The fuzzy logic of physiological cardiac hypertrophy. Hypertension 49: 962970. 2. Pluim BM, Zwinderman AH, van der Laarse A, van der Wall EE The athlete's heart. A meta-analysis of cardiac structure and function. Circulation 101: 336344. three. Barauna VG, Magalhaes FC, Krieger JE, Oliveira EM AT1 receptor participates inside the cardiac hypertrophy induced by resistance training in rats. Am J Physiol Regul Integr Comp Physiol 295: R381387. 4. Oliveira EM, Sasaki MS, Cerencio M, Barauna VG, Krieger JE Nearby renin-angiotensin program regulates left ventricular hypertrophy induced by swimming instruction independent of circulating renin: a pharmacological study. J Renin Angiotensin Aldosterone Syst 10: 1523. 5. Oliveira RS, Ferreira JC, Gomes ER, Paixao NA, Rolim NP, et al. Cardiac anti-remodelling impact of aerobic training is connected with a reduction within the calcineurin/NFAT signalling pathway in heart failure mice. J Physiol 587: 38993910. 6. Bartel DP MicroRNAs: target recognition and regulatory functions. Cell 136: 215233. 7. Lagos-Quintana M, Rauhut R, Yalcin A, Meyer J, Lendeckel W, et al. Identification of tissue-specific microRNAs from mouse. Curr Biol 12: 735739. eight. Cheng Y, Zhu P, Yang J, Liu X, Dong S, et al. Ischaemic preconditioning-regulated miR-21 protects heart against ischaemia/reperfusion injury by means of anti-apoptosis by way of its target PDCD4. Cardiovasc Res 87: 431 439. 9. Fukushima Y, Nakanishi M, Nonogi H, Goto Y, Iwai N Assessment of plasma miRNAs in congestive heart failure. Circ J 75: 336340. ten. Small EM, Olson EN Pervasive roles of microRNAs in cardiovascular biology. Nature 469: 336342. 11. Jentzsch C, Leierseder S, Loyer X, Flohrschutz I, Sassi Y, et al. A phenotypic screen to recognize hypertrophy-modulating microRNAs in principal cardiomyocytes. J Mol Cell Cardiol 52: 1320. 12. van Rooij E, Sutherland LB, Liu N, Williams AH, McAnally J, et al. A signature pattern of stress-responsive microRNAs which will evoke cardiac hypertrophy and heart failure. PNAS, USA 103: 1825518260. 13. van Rooij E, Sutherland LB, Qi X, Richardson JA, Hill J, et al. Manage of stress-dependent cardiac development and gene expression by a microRNA. Science 316: 575579. 14. Fernandes T, Hashimoto NY, Magalhaes FC, Fernandes FB, Casarini DE, et al. Aerobic workout training-induced left ventricular hypertrophy includes regulatory MicroRNAs, decreased angiotensin-converting enzyme-angiotensin ii, and synergistic regulation of angiotensin-converting enzyme 2-angiotensin. Hypertension 58: 182189. 15. LaPier TL, Swislocki AL, Clark RJ, Rodnick KJ Voluntary operating improves glucose tolerance and insulin resistance in female spontaneously hypertensive rats. Am J Hypertens 14: 708715. 16. Natali AJ, Turner DL, Harrison SM, White E Regional effects of voluntary exercise on cell size and contraction-frequency responses in rat cardiac myocytes. J Exp Biol 204: 11911199. 17. Foppa M, Duncan BB, Rohde LE Echocardiography-based left ventricular mass estimation. How should [http://www.medchemexpress.com/Calcipotriol.html MC 903] really we define hypertrophy Cardiovascular Ultrasound 3: 17. 18. McDonald JS, Milosevic D, Reddi HV, Grebe SK, Algeciras-Schimnich A Evaluation of circulating microRNA: preanalytical and analytical challenges. Clin Chem 57: 833840. 19. Sourvinou IS, Markou A, Lianidou ES Quantification of Circulating miRNAs in Plasma: Effect of Preanalytical and Analytical Para

Title Loaded From File

2017-06-27T12:59:03Z

Person8virgo:

meters on Their Isolation and Stability. [http://www.ncbi.nlm.nih.gov/pubmed/ 23388095 23388095] J Mol Diagn 15: 827834. 20. Livak KJ, Schmittgen TD Analysis of relative gene expression data using real-time quantitative PCR as well as the 2) Process. Techniques 25: 402408. 21. Lewis BP, Burge CB, Bartel DP Conserved seed pairing, generally flanked by adenosines, indicates that a large number of human genes are microRNA targets. Cell 120: 1520. 22. Huang da W, Sherman [http://www.ncbi.nlm.nih.gov/pubmed/1480666 1480666] BT, RA L Systematic and integrative evaluation of substantial gene lists using DAVID bioinformatics resources. Nat Protoc four: 4457. 23. Gentleman RC, Carey VJ, Bates DM, Bolstad B, Dettling M, et al. Bioconductor: open computer software improvement for computational biology and bioinformatics. Genome Biol 5: R80. 24. Castro MA, Wang X, Fletcher MN, Meyer KB, Markowetz F RedeR: R/ Bioconductor package for representing modular structures, nested networks and multiple levels of hierarchical associations. Genome Biol 13: R29. 25. Thum T, Gross C, Fiedler J, Fischer T, Kissler S, et al. MicroRNA-21 contributes to myocardial illness by stimulating MAP kinase signalling in fibroblasts. Nature 456: 980984. 26. Tatsuguchi M, Seok HY, Callis TE, Thomson JM, Chen JF, et al. Expression of microRNAs is dynamically regulated for the duration of cardiomyocyte hypertrophy. J Mol Cell [http://www.medchemexpress.com/Calcipotriol.html get MC 903] Cardiol 42: 11371141. 27. Zhu H, Yang Y, Wang Y, Li J, Schiller PW, et al. MicroRNA-195 promotes palmitate-induced apoptosis in cardiomyocytes by down-regulating Sirt1. Cardiovasc Res 92: 7584. 28. van Rooij E, Sutherland LB, Liu N, Williams AH, McAnally J, et al. A signature pattern of stress-responsive microRNAs that may evoke cardiac hypertrophy and heart failure. Proc Natl Acad Sci U S A 103: 1825518260. 29. Corsten MF, Dennert R, Jochems S, Kuznetsova T, Devaux Y, et al. Circulating MicroRNA-208b and MicroRNA-499 reflect myocardial harm in cardiovascular illness. Circ Cardiovasc Genet three: 499506. 30. van Rooij E, Quiat D, Johnson BA, Sutherland LB, Qi X, et al. A household of microRNAs encoded by myosin genes governs myosin expression and muscle overall performance. Dev Cell 17: 662673. 31. Duan X, Ji B, Wang X, Liu J, Zheng Z, et al. Expression of MicroRNA-1 and MicroRNA-21 in Diverse Protocols of Ischemic Conditioning in an Isolated Rat Heart Model. Cardiology 122: 3643. 32. Cheng Y, Liu X, Zhang S, Lin Y, Yang J, et al. MicroRNA-21 protects against the HO-induced injury on cardiac myocytes through its target gene PDCD4. J Mol Cell Cardiol 47: 514. 33. Patrick DM, Montgomery RL, Qi X, Obad S, Kauppinen S, et al. Stressdependent cardiac remodeling happens within the absence of microRNA-21 in mice. J Clin Invest 120: 39123916. 34. Zhang X, Azhar G, Wei JY The expression of microRNA and microRNA clusters in the aging heart. PLoS One 7: e34688. 35. Bernardo BC, Weeks KL, Pretorius L, McMullen JR Molecular distinction in between physiological and pathological cardiac hypertrophy: experimental findings and therapeutic tactics. Pharmacol Ther 128: 191227. 36. Han M, Yang Z, Sayed D, He M, Gao S, et al. GATA4 expression is primarily regulated via a miR-26b-dependent post-transcriptional mechanism in the course of cardiac hypertrophy. Cardiovasc Res 93: 645654. 37. Sayed D, Hong C, Chen IY, Lypowy J, Abdellatif M MicroRNAs play an vital function within the improvement of cardiac hypertrophy.

Use Apoptosis In A Sentence

2017-06-21T11:27:35Z

Person8virgo: Створена сторінка: [http://www.ncbi.nlm.nih.gov/pubmed/ 23388095 23388095] J Mol Diagn 15: 827834. 20. Livak KJ, Schmittgen TD Analysis of relative gene expression data using rea...

[http://www.ncbi.nlm.nih.gov/pubmed/ 23388095 23388095] J Mol Diagn 15: 827834. 20. Livak KJ, Schmittgen TD Analysis of relative gene expression data using real-time quantitative PCR and the two) Method. Strategies 25: 402408. 21. Lewis BP, Burge CB, Bartel DP Conserved seed pairing, frequently flanked by adenosines, indicates that a large number of human genes are microRNA targets. Cell 120: 1520. 22. Huang da W, Sherman [http://www.ncbi.nlm.nih.gov/pubmed/1480666 1480666] BT, RA L Systematic and integrative evaluation of significant gene lists making use of DAVID bioinformatics sources. Nat Protoc four: 4457. 23. Gentleman RC, Carey VJ, Bates DM, Bolstad B, Dettling M, et al. Bioconductor: open computer software improvement for computational biology and bioinformatics. Genome Biol 5: R80. 24. Castro MA, Wang X, Fletcher MN, Meyer KB, Markowetz F RedeR: R/ Bioconductor package for representing modular structures, nested networks and several levels of hierarchical associations. Genome Biol 13: R29. 25. Thum T, Gross C, Fiedler J, Fischer T, Kissler S, et al. MicroRNA-21 contributes to myocardial illness by stimulating MAP kinase signalling in fibroblasts. Nature 456: 980984. 26. Tatsuguchi M, Seok HY, Callis TE, Thomson JM, Chen JF, et al. Expression of microRNAs is dynamically regulated for the duration of cardiomyocyte hypertrophy. J Mol Cell Cardiol 42: 11371141. 27. Zhu H, Yang Y, Wang Y, Li J, Schiller PW, et al. MicroRNA-195 promotes palmitate-induced apoptosis in cardiomyocytes by down-regulating Sirt1. Cardiovasc Res 92: 7584. 28. van Rooij E, Sutherland LB, Liu N, Williams AH, McAnally J, et al. A signature pattern of stress-responsive microRNAs which can evoke cardiac [http://www.medchemexpress.com/MG-132.html buy MG132] hypertrophy and heart failure. Proc Natl Acad Sci U S A 103: 1825518260. 29. Corsten MF, Dennert R, Jochems S, Kuznetsova T, Devaux Y, et al. Circulating MicroRNA-208b and MicroRNA-499 reflect myocardial damage in cardiovascular disease. Circ Cardiovasc Genet three: 499506. 30. van Rooij E, Quiat D, Johnson BA, Sutherland LB, Qi X, et al. A household of microRNAs encoded by myosin genes governs myosin expression and muscle overall performance. Dev Cell 17: 662673. 31. Duan X, Ji B, Wang X, Liu J, Zheng Z, et al. Expression of MicroRNA-1 and MicroRNA-21 in Unique Protocols of Ischemic Conditioning in an Isolated Rat Heart Model. Cardiology 122: 3643. 32. Cheng Y, Liu X, Zhang S, Lin Y, Yang J, et al. MicroRNA-21 protects against the HO-induced injury on cardiac myocytes by means of its target gene PDCD4. J Mol Cell Cardiol 47: 514. 33. Patrick DM, Montgomery RL, Qi X, Obad S, Kauppinen S, et al. Stressdependent cardiac remodeling occurs inside the absence of microRNA-21 in mice. J Clin Invest 120: 39123916. 34. Zhang X, Azhar G, Wei JY The expression of microRNA and microRNA clusters within the aging heart. PLoS A single 7: e34688. 35. Bernardo BC, Weeks KL, Pretorius L, McMullen JR Molecular distinction amongst physiological and pathological cardiac hypertrophy: experimental findings and therapeutic approaches. Pharmacol Ther 128: 191227. 36. Han M, Yang Z, Sayed D, He M, Gao S, et al. GATA4 expression is mostly regulated via a miR-26b-dependent post-transcriptional mechanism throughout cardiac hypertrophy. Cardiovasc Res 93: 645654. 37. Sayed D, Hong C, Chen IY, Lypowy J, Abdellatif M MicroRNAs play an crucial role in the development of cardiac hypertrophy. Circ Res one hundred: 416 424.

Bid Apoptosis

2017-06-21T09:48:34Z

Person8virgo: Створена сторінка: that reduce steady-state levels in the [http://www.ncbi.nlm.nih.gov/pubmed/ 23388095 23388095] transcript. J Biol Chem 283: 2827428286. 39. Cheng AM, Byrom MW,...

that reduce steady-state levels in the [http://www.ncbi.nlm.nih.gov/pubmed/ 23388095 23388095] transcript. J Biol Chem 283: 2827428286. 39. Cheng AM, Byrom MW, Shelton J, Ford LP Antisense inhibition of human miRNAs and indications for an involvement of miRNA in cell growth and apoptosis. Nucleic Acids Res 33: 12901297. 40. Wu Q, Jin H, Yang Z, Luo G, Lu Y, et al. MiR-150 promotes gastric cancer proliferation by negatively regulating the pro-apoptotic gene EGR2. Biochem Biophys Res Commun 392: 340345. 41. Urbich C, Kaluza D, Fromel T, Knau A, Bennewitz K, et al. MicroRNA27a/b controls endothelial cell repulsion and angiogenesis by targeting semaphorin 6A. Blood 119: 16071616. 42. Lerner M, Lundgren J, Akhoondi S, Jahn A, Ng HF, et al. MiRNA-27a controls FBW7/hCDC4-dependent cyclin E degradation and cell cycle [http://www.medchemexpress.com/Calcipotriol.html get Calcipotriene] progression. Cell Cycle 10: 21722183. 43. Guttilla IK, White BA Coordinate regulation of FOXO1 by miR-27a, miR-96, and miR-182 in breast cancer cells. J Biol Chem 284: 2320423216. 44. Sysa-Shah P, Xu Y, Guo X, Belmonte F, Kang B, et al. Cardiac-specific over-expression of epidermal development issue receptor two induces prosurvival pathways and hypertrophic cardiomyopathy in mice. PLoS One 7: e42805. 45. McMullen JR, Shioi T, Zhang L, Tarnavski O, Sherwood MC, et al. Phosphoinositide 3-kinase plays a critical role for the induction of physiological, but not pathological, cardiac hypertrophy. Proc Natl Acad Sci U S A 100: 1235512360. 46. Reiss K, Cheng W, Ferber A, Kajstura J, Li P, et al. Overexpression of insulin-like growth factor-1 inside the heart is coupled with myocyte proliferation in transgenic mice. Proc Natl Acad Sci U S A 93: 86308635. 47. McMullen JR, Shioi T, Huang WY, Zhang L, Tarnavski O, et al. The insulin-like development factor 1 receptor induces physiological heart growth by means of the phosphoinositide 3-kinase pathway. J Biol Chem 279: 47824793. 48. Ying H, Fu H, Rose ML, McCormack AM, Sarathchandra P, et al. Genetic or pharmaceutical blockade of phosphoinositide 3-kinase p110delta prevents chronic rejection of heart allografts. PLoS One particular 7: e32892. 49. Liang Q, De Windt LJ, Witt SA, Kimball TR, Markham BE, et al. The transcription elements GATA4 and GATA6 regulate cardiomyocyte hypertrophy in vitro and in vivo. J Biol Chem 276: 3024530253. 50. Kerkela R, Kockeritz L, Macaulay K, Zhou J, Doble BW, et al. Deletion of GSK-3beta in mice leads to hypertrophic cardiomyopathy secondary to cardiomyoblast hyperproliferation. J Clin Invest 118: 36093618. 51. Li X, Kong M, Jiang D, Qian J, Duan Q, et al. MicroRNA-150 aggravates H2O2-induced cardiac myocyte injury by down-regulating c-myb gene. Acta Biochim Biophys Sin 45: 734741. 10 ~~ ~~ Fibrin D-dimer, by far the most typically made use of clinical assay of coagulation activation and in vivo fibrin formation and lysis in circulating blood has been connected with improved risk of cardiovascular illness. A meta-analysis of prospective studies showed an association of circulating levels of fibrin D-dimer with coronary heart disease that appeared of comparable strength to that of fibrinogen. Although it has been suggested that D-dimer and other fibrin degradation solutions may perhaps have a pro-inflammatory impact, the association of D-dimer and CHD threat appears independent of inflammatory markers, for instance fibrinogen, C-reactive Protein and interleukin-6.