Knot32gallon: Створена сторінка: As shown in Figure ?Figure5A,5A, 24 h Ag-NP exposures dose-dependently increased ROS generation [F(4, 13) = 28.03, p significantly increased mitochondrial ROS...

2017-04-14T12:00:35Z

Створена сторінка: As shown in Figure ?Figure5A,5A, 24 h Ag-NP exposures dose-dependently increased ROS generation [F(4, 13) = 28.03, p significantly increased mitochondrial ROS...

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As shown in Figure ?Figure5A,5A, 24 h Ag-NP exposures dose-dependently increased ROS generation [F(4, 13) = 28.03, p significantly increased mitochondrial ROS [https://en.wikipedia.org/wiki/Glafenine Glafenine] generation, as indicated by elevated levels of DCF expression [A: F(4, 13) = 28.03, p [http://www.selleckchem.com/pharmacological_MAPK.html signaling pathway] DNA damage as indicated by a significant elevated level of 8-oxo-dG production [Figure ?[Figure5B,5B, F(3, 8) = 24.78, p 2000; Wang et al., 2011). Although the mechanisms by which acetyl-L-carnitine exerts its protection are not clearly [http://www.selleckchem.com/products/E7080.html find more] defined, it works as a mitochondria protector by maintaining mitochondrial homeostasis and bioenergetics (Patel et al., 2010). In the present study, acetyl-L-carnitine effectively attenuated Ag-NP-induced oxidative damage (Figure ?(Figure5B).5B). No significant effects were observed when acetyl-L-carnitine was administered alone. The expression of the pro-apoptotic gene, Bax, was also evaluated at the protein level. Consistent with the 8-oxo-dG data, 24 h exposure of rat NSCs to 5 ��g/ml of Ag-NPs caused a significant increase in Bax protein expression [Figure ?[Figure5C,5C, F(3,8) = 6.738, p

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Knot32gallon: Створена сторінка: As shown in Figure ?Figure5A,5A, 24 h Ag-NP exposures dose-dependently increased ROS generation [F(4, 13) = 28.03, p significantly increased mitochondrial ROS...